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Nerve Agent ExposureAka: Nerve Toxin, Organophosphate Poisoning Management, Tabun, Sarin, Soman
- See Also
- Cholinergic Toxicity
- Organophosphate
- Biological Weapon
- Neurotoxin
- Pesticide
- Agents
- Tabun (GA)
- Sarin (GB)
- Soman (GD)
- GF Nerve Agent
- VX Nerve Agent
- History
- Developed in Germany before World War II
- U.S. stockpiles included GB, VX
- Iraq has used Nerve Agents frequently
- Iran-Iraq war
- Kurdish refugees
- Tokyo subway 1995
- Aum Shinrikyo cult used dilute sarin (GB)
- Pathophysiology
- Penetrates skin, eyes, and lungs
- Acetylcholinesterase inhibitors
- Most toxic of chemical agents
- Similar to insecticides (e.g. Malathion)
- Toxicity: Lethal doses
- Skin LD50 (from least to most toxic)
- GA, GB: 1000, 1700 mg
- GD, GF: 50, 30 mg
- VX: 10 mg
- Vapor LCt50 (from least to most toxic)
- GA: 400 mg-min/m3
- GB: 100 mg-min/m3
- GD: 70 mg-min/m3
- GF: Unknown
- VX: 50 mg-min/m3
- Signs: Muscarinic receptor (Smooth muscle, glands)
- Increased Glandular secretion ("All faucets on")
- Salivation
- Lacrimation
- Rhinorrhea
- Airway secretions
- Sweating
- Ocular
- Miosis
- Eye Pain
- Dim or blurred vision
- Conjunctival injection
- Respiratory
- Bronchoconstriction
- Central Apnea within minutes of severe exposure
- Gastrointestinal
- Nausea and Vomiting (early signs of liquid on skin)
- Diarrhea with severe exposure
- Signs: Nicotinic Receptors (Skeletal muscle, Ganglia)
- Skeletal Muscle changes
- Initial
- Fasciculations
- Muscle Twitching
- Later
- Weakness
- Flaccid paralysis
- Ganglionic effects
- Tachycardia
- Hypertension
- Cardiovascular Effects
- Heart Block
- Ventricular arrhythmia
- Neurologic Effects
- Onset
- Vapor: 1 minute of large exposure
- Skin contact: 1-30 minutes
- Acute
- Loss of consciousness
- Seizures
- Apnea
- Prolonged (4-6 weeks)
- Forgetfulness
- Inability to concentrate fully
- Insomnia
- Bad dreams
- Irritability
- Impaired judgment
- Depression
- No frank confusion or hallucination
- Severity of exposure (used to dose antidote)
- Vapor Exposure (Effect in seconds, Peaks in minutes)
- Low Exposure
- Miosis
- Rhinorrhea
- Dyspnea
- High Exposure
- Altered Level of Consciousness
- Seizures
- Apnea
- Flaccid Paralysis
- Death
- Liquid Exposure
- Small amount (delayed effects up to 18 hours)
- Localized sweating
- Fasciculations
- Moderate amount (delayed effects up to 18 hours)
- Gastrointestinal effects
- Large amount (Effects within 30 minutes)
- Altered Level of Consciousness
- Seizures
- Apnea
- Flaccid paralysis
- Death
- Differential Diagnosis
- Cyanide
- No increased secretions
- No Miosis
- Cyanosis uncommon
- Syncope
- Drug overdose
- Detection
- Odor
- Fish or fruit
- Detection kits
- M256A1: GB, VX (unknown threshold for GF, GA, GD)
- M272: GA, GB, GD, VX
- MINICAMS: GB, GD, VX
- ICAD: GA, GB, GD
- M18A2: GB, VX
- M21: GA, GB, GD
- M90: GA, GB, GD, GF, VX
- M93A1 Fox: GB, GD, VX
- ACAMS: GB, VX
- Bubbler: GB, VX
- CAM: GA, GB, GD, GF, VX
- DAAMS: GB, VX
- Labs: Obtain prior to therapy if possible
- Plasma Pseudocholinesterase Level
- Erythrocyte cholinesterase activity
- Severe nerve agent exposure results in <30% baseline
- Return to exposure risk when level >75% baseline
- Management
- Medical providers wear full protective gear and mask
- Level C Personal Protection Equipment
- Risk of patient off-gassing
- Decontamination
- Vapor
- Remove all clothing (risk of trapped vapor)
- Liquid
- Hypochlorite
- M291
- M258A1
- Copious water irrigation of all contacted areas
- Management: Supportive Care
- ABC Management
- Mechanical Ventilation
- Usually required for 30 minutes to 3 hours
- Airway resistance high (50-70 cm of water)
- Improves after Atropine
- Management: Emergency Department
- Atropine (Preservative-free) for pulmonary symptoms
- Adults: 2-5 mg IV q15 minutes prn pulmonary symptoms
- Child <12 years: 0.05 to 0.1 mg/kg q15 minutes prn
- Taper dose, and discontinue by 24 hours
- Pralidoxime chloride (2-PAMCl)
- Furosemide (Lasix) for pulmonary congestion
- Maximize Atropine prior to Furosemide use
- Dose: 40 to 160 mg IV prn pulmonary congestion
- Diazepam (Valium) for Seizures
- Adults: 5-10 mg IV push q5-10 minutes up to 30 mg
- Child >5 years: 0.2 to 0.5 mg/kg q5 minutes to 10 mg
- Child <5 years: 0.2 to 0.5 mg/kg q5 minutes to 5 mg
- Management: Antidotes for Military (MARK I Kits)
- Preparations
- Atropine
- Effects muscarinic receptors more than nicotinic
- Decreases secretions and improves respiratory
- MARK I Auto-injectors contain 2 mg each
- 3 kits are given to each of U.S. military
- Pralidoxime chloride (Protopam chloride, 2-PAMCl)
- Diazepam
- Raises Seizure threshold
- Useful if Status Epilepticus ensues
- Administer if 3 MARK I Kits are given at same time
- Protocol (based on severity rated above)
- Mild to moderate vapor or liquid nerve agent exposure
- Mark I kit (Atropine and 2-PAMCl): 1-2 doses
- Severe vapor or liquid nerve agent exposure
- Mark I kit: 3 doses
- Diazepam (Valium)
- Triage
- Immediate Management Indications (Antidotes)
- Severe nerve agent casualty if circulation intact
- Unconscious, convulsing, post-ictal or flaccid
- Difficult breathing or apnea
- Intact breathing and mentation with severe symptoms
- Minimal Management Indications
- Patient walking, talking with normal vital signs
- More reassuring if exposure to vapor nerve agent
- Delayed Management Indications
- Severe exposure post-stabilization
- Return to work recommendations
- Nerve agent LD50 decreases for a second exposure
- Return when cholinesterase activity >75% baseline
- Severe exposure results in <30% activity
- Severe exposure requires >45 days for recovery
- Mild to moderate exposure is less reliably predicted
- Medically observe severe exposures for >1 week
- Prevention
- Protective gear
- Chemical protective mask with Activated Charcoal
- Charcoal in chemical protective over-garment
- Butyl rubber in chemical protective gloves and boots
- Antidotal Enhancement ("Pretreatment")
- Pyridostigmine Bromide 30 mg PO q8h before exposure
- Research topics
- Seizure protection
- More reliable alternatives to Valium (e.g. Versed)
- Neuro-protective agents
- Nerve agent antidotes
- Circulating bio-scavengers for nerve agent
- Complications: Long-term
- Neuropsychiatric changes may persist weeks to months
- Polyneuropathy
- Cognitive changes associated with prolonged Seizures
- References
- Medical Response to Chemical Warfare and Terrorism
- US Army Medical Research Institute Chemical Defense
- Video-Teleconference: 4/20/00 to 4/22/99
- Video-Teleconference: 12/5/00 to 12/7/00
- Text: 3rd Edition, December 1998
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| Definition (MSH) | An organophosphorus ester compound that produces potent and irreversible inhibition of cholinesterase. It is toxic to the nervous system and is a chemical warfare agent. |
| Concepts | Organophosphorus Compound (T115)
, Hazardous or Poisonous Substance (T131)
|
| English | Isopropyl methylphosphonofluoridate, Nerve agent GB, o Isopropylmethyl Phosphonofluoridate, ortho Isopropylmethyl Phosphonofluoridate, Sarin |
| Spanish | sarina |
| Credits | Derived from the NIH UMLS (Unified Medical Language System)
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|
|---|
| Definition (MSH) | An organophosphorus compound that inhibits cholinesterase. It causes seizures and has been used as a chemical warfare agent. |
| Concepts | Organophosphorus Compound (T115)
, Pharmacologic Substance (T121)
, Hazardous or Poisonous Substance (T131)
|
| English | Nerve agent GD, Pinacolyl Methylphosphonofluoridate, Soman |
| Credits | Derived from the NIH UMLS (Unified Medical Language System)
|
|
|---|
| Concepts | Organophosphorus Compound (T115)
, Hazardous or Poisonous Substance (T131)
|
| English | Nerve agent GA, tabun |
| Credits | Derived from the NIH UMLS (Unified Medical Language System)
|
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