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RofecoxibAka: Vioxx
- See Also
- Precaution
- Vioxx voluntarily withdrawn from market October 2004
- Withdrawal due to increased risk of MI and CVA
- Vioxx voluntarily withdrawn from market October 2004
- Class
- NSAID: COX2 Inhibitor
- Similar to Celecoxib (Celebrex)
- Advantages
- Dosing
- Osteoarthritis: 12.5 to 25 mg PO qd
- Acute pain or Dysmenorrhea: 50 mg PO qd
- Pharmacokinetics
- Oral Bioavailability: 92%
- Peaks: 2-3 hours (delayed by food intake)
- Half life: 17 hours
- Metabolized by hepatic reduction
- Factors increasing serum concentrations
- Age over 65 years old
- Hepatic insufficiency
- Efficacy: Equivalent analgesia to 50 mg Vioxx
- Drug Interactions
- Decreased Vioxx serum concentrations
- Antacids (decrease Vioxx serum concentrations 20%)
- Rifampin (decrease Vioxx serum concentrations 50%)
- Increased concentrations of other medications
- Raises serum Methotrexate levels 23%
- Raises Warfarin levels - increases ProTime 10%
- Decreased Vioxx serum concentrations
- Adverse Effects
- See COX-2 Inhibitor for Nonfatal MI risk
- Confirmed in larger trial and withdrawn from market
- Diarrhea
- Nausea
- Dyspepsia
- Abdominal Pain
- Lower extremity edema
- Increased Blood Pressure
- See COX-2 Inhibitor for Nonfatal MI risk
- References
rofecoxib (C0762662) | |
|---|---|
| Definition (NCI) | A substance used for pain relief that is also being studied for its ability to prevent cancer and to block the growth of new blood vessels to solid tumors. It belongs to the family of drugs called nonsteroidal anti-inflammatory drugs. |
| Definition (PDQ) | A synthetic, nonsteroidal derivative of phenyl-furanone with antiinflammatory, antipyretic and analgesic properties and potential antineoplastic properties. Rofecoxib binds to and inhibits the enzyme cyclooxygenase-2 (COX-2), resulting in an inhibition of the conversion of arachidonic acid to prostaglandins. COX-related metabolic pathways may represent key regulators of cell proliferation and neo-angiogenesis. Some epithelial tumor cell types overexpress pro-angiogenic COX-2. Check for "http://www.cancer.gov/Search/ClinicalTrialsLink.aspx?id=38568&idtype=1" active clinical trials or "http://www.cancer.gov/Search/ClinicalTrialsLink.aspx?id=38568&idtype=1&closed=1" closed clinical trials using this agent. ("http://nciterms.nci.nih.gov:80/NCIBrowser/ConceptReport.jsp?dictionary=NCI_Thesaurus&code=C1832" NCI Thesaurus) |
| Definition (NCI) | A synthetic, nonsteroidal derivative of phenyl-furanone with antiinflammatory, antipyretic and analgesic properties and potential antineoplastic properties. Rofecoxib binds to and inhibits the enzyme cyclooxygenase-2 (COX-2), resulting in an inhibition of the conversion of arachidonic acid to prostaglandins. COX-related metabolic pathways may represent key regulators of cell proliferation and neo-angiogenesis. Some epithelial tumor cell types overexpress pro-angiogenic COX-2. (NCI04) |
| Concepts | Organic Chemical (T109) , Pharmacologic Substance (T121) |
| MSH | C116926 |
| English | rofecoxib, ROFECOXIB PREPARATION, Rofecoxib product |
| Spanish | rofecoxib |
| Parent Concepts | Cyclooxygenase Inhibitors (C0085387), Lactones (C0022947), [MS102] NONSALICYLATE NSAIS, ANTIRHEUMATIC (C0973575), Cyclooxygenase 2 Inhibitors (C1257954), Duplicate concept (C1274013) |
| Sources | CSP, MSH, NCI, NDFRT, PDQ, RXNORM, SCTSPA, SNOMEDCT, VANDF Derived from the NIH UMLS (Unified Medical Language System) |
Vioxx (C0876768) | |
|---|---|
| Concepts | Organic Chemical (T109) , Pharmacologic Substance (T121) |
| MSH | C116926 |
| English | Cahill May Roberts brand of rofecoxib, Merck brand of rofecoxib, Merck Frosst brand of rofecoxib, Merck Sharp & Dhome brand of rofecoxib, MSD brand of rofecoxib, Vioxx, Vioxx Dolor |
| Sources | CSP, MSH, NCI, PDQ, RXNORM Derived from the NIH UMLS (Unified Medical Language System) |