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Tacrolimus OintmentAka: Protopic, Elidel, Pimecrolimus
- Indications
- Severe refractory Atopic Dermatitis
- Pharmacokinetics
- Normal topical use causes <5 ng/ml serum concentration
- Greater absorption in severely damaged skin
- Factors increasing serum concentrations
- Higher doses or greater surface area of application
- Inflamed skin
- Mechanism
- Macrolide with similar activity to cyclosporine
- Specifically binds T Cell receptors
- Inhibits cytokine production
- Inhibits IgE sensitivity
- Precautions
- Increased risk of skin malignancy and Lymphoma
- FDA black box warning has been added
- Do not use for extended periods of time
- Use only in refractory eczema
- Preparations
- Pimecrolimus (Elidel)
- Tacrolimus (Protopic)
- Dosing: Tracolimus
- Children over age 2 years: Apply 0.03% ointment bid
- Adults: Apply 0.03% or 0.1% ointment bid
- Adverse Effects
- Local irritation (transient <1 week)
- Burning
- Erythema
- Pruritus
- Less common adverse effects
- Skin Tingling
- Acne
- Hyperesthesia
- Alcohol intolerance
- Immunosuppression related adverse effects
- Varicella zoster increased Incidence
- Eczema herpeticum
- Increased malignancy risk in mice (see precautions)
- Skin cancer
- Lymphoma
- Precautions
- Protect treated areas from direct sunlight
- Systemic tacrolimus associated with skin cancer
- Non-Melanoma skin cancer
- Advantages
- Very effective in refractory atopic cases
- Unlike Corticosteroids, does not cause skin atrophy
- May be useful on face and neck
- Disadvantages
- Expensive (costs per Red Book)
- Tacrolimus ointment (0.03 or 0.1%): $60 per 30 grams
- Risk of malignancy (see prcautions)
- Skin cancer
- Lymphoma
- References
- Bekersky (2001) J Am Acad Dermatol 44:S17
- Hanifin (2001) J Am Acad Dermatol 44:S28
- Kang (2001) J Am Acad Dermatol 44:S58
- Paller (2001) J Am Acad Dermatol 44:S47
- Ruzicka (1997) N Engl J Med 337:816
- (2001) Med Lett Drugs Ther 43(1102):33
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| Concepts | Organic Chemical (T109)
, Pharmacologic Substance (T121)
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| English | Elidel, Novartis brand of pimecrolimus |
| Credits | Derived from the NIH UMLS (Unified Medical Language System)
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| Definition (NCI) | A 33-epi-chloro-derivative of the ascomycin macrolactam with immunosuppressant property. Pimecrolimus binds to the receptor macrophilin-12 (FKBP-12) forming a complex that blocks the calcium-dependent signal transduction cascade mediated by calcineurin. Via dephosphorylation, calcineurin is the enzyme responsible for activating nuclear factor of activated T-cells (NF-AT), a T cell transcriptional regulatory factor. As a consequence, the synthesis and release of Th1- (T helper 1) and Th2- (T helper 2) type cytokines, and other inflammatory mediators from T-cells and mast cells are blocked and the expression of signals essential for the activation of inflammatory T-lymphocytes is inhibited. However, pimecrolimus mode of action is cell-selective and does not affect Langerhans' cells/dendritic cells and primary fibroblasts. |
| Concepts | Organic Chemical (T109)
, Pharmacologic Substance (T121)
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| English | Pimecrolimus, PIMECROLIMUS @ @ UNIDENTIFIED, PIMECROLIMUS PREPARATION, PIMECROLIMUS UNIDENTIFIED |
| Spanish | pimecrolimo, pimecrolimus |
| Credits | Derived from the NIH UMLS (Unified Medical Language System)
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